Who gets Hemochromatosis

Who Gets Hemochromatosis?and maintain that level. Other comorbid conditions
Hereditary hemochromatosis (HH) is the most(conditions associated with HH) must be looked
common form of hemochromatosis. It isfor and treated. Of especial importance is
predominately a disorder of persons of northerndiabetes. The patients family should also be
European extraction. In this form ofscreened for HH. Those at greatest risk are the
hemochromatosis patients are most commonlypatients siblings. However all first degree relatives
homozygous for (carry two copies of) the C282Yshould be screened. Initial testing consists of
mutation of the HFE gene. Sometimes patientsferritin levels, transferrin saturations and genetic
may carry one copy of the C282Y mutation andtesting. In this way many early cases of HH are
one copy of the H63D mutation. There are othernow being picked up and successfully treated
forms of hemochromatosis and iron overloadbefore patients load enough iron to give the
which will be discussed later.severe consequences of organ failure.
Hereditary hemochromatosis takes many yearsWhat Happens If The Patient Cannot Tolerate
to display its true nature. This is because it takesVenesections?
time to load iron in the body. Men usually loadOur patient who we shall call Mrs. Abbott is a
more quickly than women. This is becausesmall lady. She weighs in at just over 110 lbs and
women have monthly menstrual cycles and haveis only 5 foot 2 inches tall. After Mrs. Abbotts first
children. Each pregnancy is equivalent to the lossvenesection of 500 mls (which is equivalent to
of 1 gram of iron. So the bar is set lower to250 mcg of iron) she is totally exhausted and has
confirm the diagnosis of HH in women. Before thedifficulty standing for some three days. This is
true genetic nature of the disorder wasdespite adequate hydration before and after
recognized the diagnosis was made if the patientvenesection. This lady is also known to have
could be venesected or deironed of 5 grams ofosteoporosis (the rate of which is increased in HH)
blood (if male) without causing significant anemia.so it is important that she not fall as she could
If the patient was a woman only 3 grams of ironeasily break an arm or worse a hip. Another
had to be removed by venesection withoutvenesection is planned for a week later.
causing significant anemia to make the diagnosis.What needs to be done is to decrease the
Liver biopsy was not infrequently employed tostandard venesection unit down to 250 mls and
confirm the diagnosis.probably do this every few weeks after she has
Now with the ready availability of genetic testingadequately recovered. It will take longer to deiron
the criteria are different. Often the diagnosis canMrs. Abbott, but her treatment overall is safer. If
be made using genetic testing and iron studies.Mrs. Abbott had a ferritin level of say 2,000 ug/L
Liver biopsy is often now not necessary.then it would be a tougher call. She would need to
What Happens When A Patient Presents?be deironed more quickly.
Let us assume that a patient has presented withImmediately after venesection a cold pack was
fatigue and arthralgia (aching joints) to a doctor.applied to Mrs. Abbotts veins. This was done to
Now many things can give such a presentation.preserve the veins. This is especially important in
Paradoxically one of them is anemia or ironpersons with frail veins and those who will need
deficiency. Anyhow let us assume that the patientmultiple venesections.
is suspected of having hemochromatosis. TheAfter a further 20 venesections of only 250 mls
patient is a lady of some 65 years of age. Sheeach Mrs. Abbott has a ferritin level of 46 ug/L.
had five children and menopause at age 45 years.She has been successfully deironed. Her liver
She is of Irish/Scottish extraction and her motherfunction tests are now normal and her transferrin
died of liver problems yet her mother neversaturation has fallen to 45%. Proper treatment is
drank a drop of alcohol in her life. The patient isto now monitor Mrs. Abbott and see how quickly
quite fair skinned. However this may be a redshe loads iron. She is probably a patient who may
herring as not all patients with hemochromatosisonly require 2 or 3 (half) venesections per year.
go a bronzed or grayish color. When the patient isWhy Is It Important To Screen Relatives?
examined by the doctor she is noted to haveNow Mrs. Abbott is from a very large family. She
enlarged and painful second and third knuckles andhas 10 siblings, all of whom are younger and still
pain at the base of her thumbs. This is known asalive. There are 5 sisters and 5 brothers. All the
iron fist and is a clue to hemochromatosis.siblings are willing to be tested and live close.
Examination of the patients abdomen reveals anOften people simply do not want to know. This is
enlarged liver. So preliminary testing is done. Thisof course their right.
patient is found to have an enlarged liver onBecause one C282Y mutation is inherited from
ultrasound but no cirrhosis. Her ferritin level is 650each parent Mrs. Abbotts parents were at least
ug/L and she has a transferrin saturation of 96%.carriers of one C282Y mutation or were C282Y
In itself these are big clues this patient probablyheterozygotes. Both her parents are deceased so
has HH. Genetic testing reveals the patient to bethey cannot be tested. If we assume that both
a carrier of two copies of C282Y. Liver functionparents were C282Y heterozygotes then the
tests are slightly abnormal. Now this patient haschance of each of their children carrying two
HH. A liver biopsy is probably not necessary ascopies of C282Y is 1 in 4 or 25%. If one parent
the ferritin level is not markedly elevated.was a C282Y homozygote or carried two copies
Interestingly this patient almost certainly hasof C282Y then the children have a 50% chance
fibrosis of the liver which may well be reversed ifof carrying two copies of C282Y. If both Mrs.
the patient is properly deironed.Abbotts parents carried two copies of C282Y
The treatment for this patient is to deiron herthen all her siblings will carry two copies of C282Y.
down to a ferritin level of 25 to 75 ug/L and try